Trimethoxy Cinnamoyl Reserpate De Methyl

Trimethoxy Cinnamoyl Reserpate De Methyl Uses, Dosage, Side Effects, Food Interaction and all others data.

Trimethoxy Cinnamoyl Reserpate De Methyl is an angiotensin-converting enzyme inhibitor used as an antihypertensive drug. It is an alkaloid obtained from Rauwolfia serpentina and other species of Rauwolfia.

Used to treat hypertension. Trimethoxy Cinnamoyl Reserpate De Methyl inhibits angiotensin-converting enzyme. ACE is a peptidyl dipeptidase that catalyzes the conversion of angiotensin I to the vasoconstrictor substance, angiotensin II. Angiotensin II also stimulates aldosterone secretion by the adrenal cortex and general vasoconstriction, both of which lead to increases vascular resistance. By inhibiting angiotensin II, aldosterone reabsorption is decreased as well as vasoconstriction. This combined effect serves to decrease blood pressure.

Table Of contents

• Trimethoxy Cinnamoyl Reserpate De Methyl
• Uses
• Dosage
• Side Effect
• Precautions
• Interactions
• Uses during Pregnancy
• Uses during Breastfeeding
• Accute Overdose
• Food Interaction
• Half Life
• Volume of Distribution
• Clearance
• Interaction With other Medicine
• Contradiction
• Storage

Trade Name	Trimethoxy Cinnamoyl Reserpate De Methyl
Generic	Rescinnamine
Rescinnamine Other Names	Rescinnamine, Trimethoxy cinnamoyl reserpate de methyl
Type
Formula	C35H42N2O9
Weight	Average: 634.716 Monoisotopic: 634.289030952
Groups	Approved
Therapeutic Class
Manufacturer
Available Country
Last Updated:	September 19, 2023 at 7:00 am

Uses

For the treatment of hypertension.

How Trimethoxy Cinnamoyl Reserpate De Methyl works

Trimethoxy Cinnamoyl Reserpate De Methyl Binds to and inhibits the angiotensin converting enzyme. Trimethoxy Cinnamoyl Reserpate De Methyl competes with angiotensin I for binding at the angiotensin-converting enzyme, blocking the conversion of angiotensin I to angiotensin II. Inhibition of ACE results in decreased plasma angiotensin II. As angiotensin II is a vasoconstrictor and a negative-feedback mediator for renin activity, lower concentrations result in a decrease in blood pressure and stimulation of baroreceptor reflex mechanisms, which leads to decreased vasopressor activity and to decreased aldosterone secretion.

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